Archive for the ‘Birth Injury Law’ Category

Amniotic Fluid Embolism Syndrome

Thursday, October 20th, 2011

labor delivery malpractice Amniotic Fluid Embolism SyndromeAmniotic fluid embolism syndrome (also known as anaphylactoid syndrome of pregnancy) is a disastrous complication of pregnancy and childbirth. The condition can be triggered by unpreventable effects of labor and delivery, or by medical malpractice at birth.

For an amniotic fluid embolism to occur, three conditions must be present:

  • (1) ruptured membranes of the amniotic sac (a “broken water”)
  • (2) ruptured uterine or cervical veins
  • (3) a pressure gradient from uterus to vein

When and amniotic fluid embolism occurs, amniotic fluid enters the mother’s circulatory system via the site of a uterine trauma (such as a laceration), the placental insertion site, or via endocervical veins, which can occur due to childbirth trauma, abortion, abdominal trauma during pregnancy, or even procedures such as an amniotic fluid infusion. This amniotic fluid may contain fetal squamous cells, fetal mucin, fetal hair, or other fetal “debris,” which can then migrate to the mother’s heart, lungs, brain, kidneys, liver, spleen, and pancreas. The amniotic fluid can trigger inflammation, allergic reaction, collapse of the heart and lungs, shock, or respiratory failure.

Amniotic fluid embolism syndrome occurs very infrequently. In fact, only one patient suffers an amniotic fluid embolism for every 8,000 to 80,000 births. Yet for some reason as-yet unknown, the incidence of amniotic fluid embolisms in the U.S. is on the rise.

And when an amniotic fluid embolism occurs, the results are extreme. First, the victim begins having shortness of breath and very high blood pressure. These symptoms rapidly proceed to cardiac or cardiopulmonary arrest or coma. This is known as the “first phase” of amniotic fluid embolism.

For those victims who survive the first phase (around forty percent of victims), a second phase follows. The second phase of amniotic fluid embolism, sometimes called the hemorrhagic phase, generally involves coughing, vomiting, severe chills and shivering, excessive bleeding, and, if the victim is conscious, a bad taste in her mouth. For those women who have not yet given birth, the reduction of oxygen supply during both phases can lead to fetal distress and danger.

Roughly half of all women who suffer an amniotic fluid embolism die within an hour after their symptoms begin. Overall, the death rate for victims of amniotic fluid embolism is around 80 percent, although around 70 percent of infants survive. Of those women who survive an amniotic fluid embolism, a large majority are left with permanent brain damage.

There are certain risk factors which help to predict amniotic fluid embolisms before they occur. For example, sudden or violent labor, advanced maternal age, delivery via C-section or using forceps or vacuum extraction, placenta previa, placental abruption, cervical lacerations, eclampsia, and enduced labor are all risk factors. Patients who are critically ill, or who suffer from venous thromboembolism, are at particular risk.  The failure to anticipate and protect against amniotic fluid embolism in patients who have one or more risk factors may itself constitute medical negligence.

If you or a loved one has suffered an amniotic fluid embolism, talk to an experienced medical malpractice attorney. Your attorney, working with expert medical advisers, can help you to determine the causes of the amniotic fluid embolism in your case, and help you to decide whether to take legal action.

If you have any questions about a serious injury resulting from a medical procedure, please give us a call us at 312-527-4500 or email us at info@passenlaw.com for a complimentary consultation. You can also learn more by following us on Twitter, reviewing our LinkedIn or Avvo.com pages, and by reviewing our website.

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Repeat C-Section vs. Vaginal Birth After Cesarean

Thursday, September 29th, 2011

c section delivery 300x242 Repeat C Section vs. Vaginal Birth After CesareanThere has been much controversy in recent years over the use of VBAC (or vaginal birth after cesarean) as opposed to repeat cesarean sections (“C-section”) for mothers who have previously delivered via c-section. In the past, it was believed that a woman who delivered a baby by cesarean delivery would require a C-section for all future deliveries. We now know this is not always the case.

Each method of delivery carries its own risks and benefits and, unsurprisingly, the decision as to which method should be used often depends on highly individual factors.  Doctors who adhere blindly to one method or the other thus risk injuring, or even killing, mothers and infants who are best suited to another choice. The failure to properly analyze each case and select the best option for the mother can thus constitute birth injury malpractice.

Cesarean sections, or c-sections, are surgical births. When a c-section is performed, a doctor cuts a hole into the mother’s abdomen, reaches inside, and manually delivers an infant. It does not take a medical expert to know that such a procedure carries risks. Among those are the risks associated with all surgeries, including possible infection,  scar tissue, blood clots or pulmonary embolism, and anesthesia complications. C-sections also increase the risk that repeat c-sections or hysterectomy will be required.

Vaginal birth after cesarean, also called a trial of labor after cesarean delivery (TOLAC) is, just as it sounds, a natural childbirth following a delivery by c-section. Whatever choice is made can be dangerous if doctors and hospital staff are not properly trained and careful.

The benefits of a VBAC may include the following:

  • Reduced risk of thromboembolism (blood clot in the leg or lung)
  • Shorter length of hospital stay in most cases
  • Less likely to need a blood transfusion
  • Possible lower rate of postpartum fever, wound infection, uterine infection
  • Fewer neonatal breathing problems

The primary reason that many women select VBAC is to avoid a second c-section, invasive abdominal surgery. The risks of injury at birth, both to the mother and infant, increase with each subsequent c-section, and with each year that the mother ages.

But attempts at VBAC are successful only 70-80% of the time. In the remaining 20-30% of VBAC attempts, a c-section is needed after the attempt, often on an emergency basis.

The greatest risk associated with VBAC is the risk of uterine rupture. Just as it sounds, a uterine rupture occurs when the uterus tears open during childbirth contractions, almost always at the site of the previous internal c-section scar. Because of the weakness of the c-section scar, uterine ruptures are more than twice as likely in VBAC births than ordinary vaginal births.

Still, less than 1% of VBAC attempts end in uterine rupture. But when ruptures do occur, the consequences are severe. One in twenty infants die in a delivery where uterine rupture occurs. The mothers themselves then have a higher risk of infection.

There are many factors, however, which influence the risk of uterine rupture in specific cases. For instance, if chemical induction of labor through pitocin is used (or labor is augmented in this fashion), the risk of uterine rupture increases by fifteen times. Indeed, our attorneys believe that the use of pitocin during a VBAC attempt is clear evidence of medical negligence at delivery.

Likewise, the type of incision used in the prior c-section greatly influences the risk of uterine rupture in an attempted VBAC. There are three c-section incision types: the classic incision, made high on the abdomen and vertically, the low vertical incision, and the low transverse incision (often called the “bikini” c-section). Either low incision carries a substantially lower risk of uterine rupture during VBAC.

There are numerous other factors which also influence VBAC risk, including the size of the mother’s pelvis, the reason or reasons for the prior c-section and the size and position of the baby. To avoid medical malpractice, a doctor must consider each of these factors before advising a patient on the decision between repeat c-section and VBAC.

So, why do many physicians and hospitals discourage or prohibit VBAC attempts, regardless of the individual factors? It appears that the culprit is physician convenience. Since 2004, the American College of Obstetricians and Gynecology (ACOG), the governing organization of virtually all U.S. obstetricians, has stated in its guidelines that when VBAC is attempted, the doctor and anesthesiologist must be immediately available – meaning present at the hospital throughout labor.

ACOG further states that VBAC is an acceptable option for women who:

  • Do not have other conditions (as an example, placenta previa) that require cesarean delivery
  • Have only one low transverse uterine incision from a past cesarean delivery
  • Have no other uterine scars and has never experienced a uterine rupture
  • Do not have pelvic problems or abnormalities that prevent vaginal delivery
  • Have a baby in the proper position (head down)

Where such factors are not present, vaginal delivery should not be attempted; to do so may be considered medical malpractice.  If you are pregnant after a previous c-section, be sure to speak to your physician about whether VBAC or repeat c-section is best in your particular case, and ask about the factors discussed above.  Only once your individual situation is analyzed can you make the best, and safest, choice for you and your baby.

If you have any questions about a birth injury or medical malpractice matter, please give us a call us at 312-527-4500 or email us at info@passenlaw.com for a complimentary consultation. You can also learn more by following us on Twitter, reviewing our LinkedIn or Avvo.com pages, and by reviewing our website.

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Promising Research on Fetal Brain Injuries

Friday, September 16th, 2011

The birth injury attorneys of Passen Law Group have often written on the various brain injuries and permanent conditions associated with fetal oxygen deprivation. Whether due to a condition during pregnancy, birth trauma, medical malpractice, or some combination of these factors, the consequences of fetal brain damage are devastating to the infant and her parents. Injuries from fetal oxygen deprivation include:

•    epilepsy;
•    cerebral palsy;
•    mental retardation;
•    autism; and
•    schizophrenia.

Now, new research offers some hope for the successful treatment of these injuries in the future. Researchers from The Scripps Research Institute studying fetal oxygen deprivation in experiments on mice have found that brain injury from such deprivation is tied to the action of lysophosphatidic acid , or LPA, a particular fatty molecule which functions as a receptor transferring information into developing brain cells.

These researchers believe that their findings point to LPA as a key factor in injury developing when a fetus is deprived of oxygen. While the research is not far enough yet to even confirm the role of LPA, let alone design an effective treatment, this finding is promising indeed.

LPA normally acts as a signal to assist in the development of the human brain – both in utero and after birth. The chemical signals from LPA inflence neurogenesis, the process by which new neurons are formed in the fetal brain and the architecture of the brain is constructed.

As LPA signals the formation of new neurons, new areas of the brain form rapidly. Some of these areas are essential for survival outside the womb – for instance, the portions of the brain controlling breathing, drinking, digestion, and other basic functions. Others continue to develop after the infant enters the outside world.

To the layperson, it seems self-evident that oxygen deprivation leads to brain damage. But research leader and Scripps Research Professor Jerold Chun, MD, PhD said that his team’s research indicated that it is not the lack of oxygen itself which triggers damage in the very young, but the chemical changes which occur in the brain are actually intimately tied to the brain’s reactions to LPA.

When an fetus suffers hypoxia (oxygen deprivation), neurons in the brain become overstimulated, as they would if exposed to excessive levels of LPA. When the researchers either genetically removed LPA receptors in the subject mice, or blocked these receptors using medication, the fetal mice could suffer hypoxia without suffering brain damage.

If further research confirms and expands on this recent discovery, it is possible that infants who suffer oxygen deprivation could be successfully treated with drugs to prevent LPA from triggering the changes in the brain which turn deprivation into permanent injury and disability. This is particularly exciting as there is currently no known treatment for the brain injury caused by fetal oxygen deprivation.

While it is too soon to know whether the results achieved in mice can be replicated in humans, the brain injury lawyers of Passen Law Group are encouraged by this promising line of research. We hope that, in time, the suffering of the many infants who suffer from fetal oxygen deprivation, and that of their parents, can be mitigated or eliminated.

The research appeared in an advance, online issue of the journal Proceedings of the National Academy of Sciences (PNAS).

For a free consultation with an experienced Chicago birth injury lawyer at Passen Law Group, call us at (312) 527-4500.

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